1 |
Leoni D, Rello J. Cardiac arrest among patients with infections: causes, clinical practice and research implications[J]. Clin Microbiol Infect, 2017, 23 (10): 730-735.
|
2 |
Ni J, Lin M, Jin Y, et al. Gas6 attenuates sepsis-induced tight junction injury and vascular endothelial hyperpermeability via the Axl/NF-κB signaling pathway[J]. Front Pharmacol, 2019 (10): 662.
|
3 |
Liu Y, Mu S, Li X, et al. Unfractionated heparin alleviates sepsis-induced acute lung injury by protecting tight junctions[J]. J Surg Res, 2019 (238): 175-185.
|
4 |
Cheng Z, Qi R, Li L, et al. Dihydroartemisinin ameliorates sepsis-induced hyperpermeability of glomerular endothelium via up-regulation of occludin expression[J]. Biomed Pharmacother, 2018 (99): 313-318.
|
5 |
Weng J, Yu L, Chen Z, et al. β-Catenin phosphorylation at Y654 and Y142 is crucial for high mobility group box-1 protein-induced pulmonary vascular hyperpermeability[J]. J Mol Cell Cardiol, 2019 (127): 174-184.
|
6 |
Ince C, Mayeux PR, Nguyen T, et al. The endothelium in sepsis[J]. Shock, 2016, 45 (3): 259-270.
|
7 |
Xiao F, Wang D, Kong L, et al. Intermedin protects against sepsis by concurrently re-establishing the endothelial barrier and alleviating inflammatory responses[J]. Nat Commun, 2018, 9 (1): 2644.
|
8 |
Delvaeye T, De Smet MAJ, Verwaerde S, et al. Blocking connexin43 hemichannels protects mice against tumour necrosis factor-induced inflammatory shock[J]. Sci Rep, 2019, 9 (1): 16623.
|
9 |
Murphy TV, Sandow SL. Agonist-evoked endothelial Ca2+ signalling microdomains[J]. Curr Opin Pharmacol, 2019 (45): 8-15.
|
10 |
Leybaert L, Lampe PD, Dhein S, et al. Connexins in cardiovascular and neurovascular health and disease: pharmacological implications[J]. Pharmacol Rev, 2017, 69 (4): 396-478.
|
11 |
Lee W, Lee H, Lee T, et al. Inhibitory functions of maslinic acid, a natural triterpene, on HMGB1-mediated septic responses[J]. Phytomedicine, 2020 (69): 153200.
|
12 |
Park JY, Lee HJ, Kwon YS, et al. 3,4,5-trihydroxycinnamic acid inhibits LPS-induced inflammatory response by increasing SIRT1 expression in human umbilical vein endothelial cells[J]. J Vasc Res, 2020, 57 (5): 302-310.
|
13 |
Hauschildt J, Schrimpf C, Thamm K, et al. Dual pharmacological inhibition of angiopoietin-2 and VEGF-A in murine experimental sepsis[J]. J Vasc Res, 2020, 57 (1): 34-45.
|
14 |
Luo L, Xu M, Liao D, et al. PECAM-1 protects against DIC by dampening inflammatory responses via inhibiting macrophage pyroptosis and restoring vascular barrier integrity[J]. Transl Res, 2020 (222): 1-16.
|
15 |
李爽,程波,王强,等.血小板与内皮细胞屏障功能[J/CD].中华危重症医学杂志(电子版),2019,12(1):56-60.
|
16 |
McDonnell CJ, Garciarena CD, Watkin RL, et al. Inhibition of major integrin αVβ3 reduces Staphylococcus aureus attachment to sheared human endothelial cells[J]. J Thromb Haemost, 2016, 14 (12): 2536-2547.
|
17 |
Schmidt EP, Overdier KH, Sun X, et al. Urinary glycosaminoglycans predict outcomes in septic shock and acute respiratory distress syndrome[J]. Am J Resp Crit Care, 2016, 194 (4): 439-449.
|
18 |
Song JW, Zullo JA, Liveris D, et al. Therapeutic restoration of endothelial glycocalyx in sepsis[J]. J Pharmacol Exp Ther, 2017, 361 (1): 115-121.
|
19 |
Lee YH, Park JH, Cheon DH, et al. Processing of syndecan-2 by matrix metalloproteinase-14 and effect of its cleavage on VEGF-induced tube formation of HUVECs[J]. Biochem J, 2017, 474 (22): 3719-3732.
|
20 |
Chao CH, Chen HR, Chuang YC, et al. Macrophage migration inhibitory factor-induced autophagy contributes to thrombin-triggered endothelial hyperpermeability in sepsis[J]. Shock, 2018, 50 (1): 103-111.
|
21 |
Li Z, Yin M, Zhang H, et al. BMX represses thrombin-PAR1-mediated endothelial permeability and vascular leakage during early sepsis[J]. Circ Res, 2020, 126 (4): 471-485.
|
22 |
Zeng Z, Yang Y, Dai X, et al. Polydatin ameliorates injury to the small intestine induced by hemorrhagic shock via SIRT3 activation-mediated mitochondrial protection[J]. Expert Opin Ther Targets, 2016, 20 (6): 645-652.
|
23 |
Wu J, Deng Z, Sun M, et al. Polydatin protects against lipopolysaccharide-induced endothelial barrier disruption via SIRT3 activation[J]. Lab Invest, 2020, 100 (4): 643-656.
|
24 |
Bentzer P, Fisher J, Kong HJ, et al. Heparin-binding protein is important for vascular leak in sepsis[J]. Intensive Care Med Exp, 2016, 4 (1): 33.
|
25 |
Rasmuson J, Kenne E, Wahlgren M, et al. Heparinoid sevuparin inhibits Streptococcus-induced vascular leak through neutralizing neutrophil-derived proteins[J]. FASEB J, 2019, 33 (9): 10443-10452.
|
26 |
Deng M, Tang Y, Li W, et al. The endotoxin delivery protein HMGB1 mediates caspase-11-dependent lethality in sepsis[J]. Immunity, 2018, 49 (4): 740-753.e7.
|
27 |
孙雪东,严一核,褚韦韦,等.高迁移率族蛋白B1/Toll样受体4信号通路在脓毒症大鼠致急性肺损伤中的作用研究[J/CD].中华危重症医学杂志(电子版),2020,13(6);419-426.
|
28 |
陈敏,袁佳辉,郭冬阳,等. CXC趋化因子配体12/CXC趋化因子受体4轴在脓毒症中的研究进展[J/CD].中华危重症医学杂志(电子版),2020,13(3):225-228.
|
29 |
Geven C, Peters E, Schroedter M, et al. Effects of the humanized anti-adrenomedullin antibody adrecizumab (HAM8101) on vascular barrier function and survival in rodent models of systemic inflammation and sepsis[J]. Shock, 2018, 50 (6): 648-654.
|
30 |
Zheng Q, Wang YC, Liu QX, et al. FK866 attenuates sepsis-induced acute lung injury through c-jun-N-terminal kinase (JNK)-dependent autophagy[J]. Life Sci, 2020 (250): 117551.
|
31 |
Stober VP, Lim YP, Opal S, et al. Inter-α-inhibitor ameliorates endothelial inflammation in sepsis[J]. Lung, 2019, 197 (3): 361-369.
|
32 |
中国医疗保健国际交流促进会急诊医学分会,中华医学会急诊医学分会,中国医师协会急诊医师分会,等.中国脓毒症早期预防与阻断急诊专家共识[J].中华急诊医学杂志,2020,29(7):885-895.
|
33 |
Kuck JL, Bastarache JA, Shaver CM, et al. Ascorbic acid attenuates endothelial permeability triggered by cell-free hemoglobin[J]. Biochem Biophys Res Commun, 2018, 495 (1): 433-437.
|
34 |
李娜,王美霞,周晋萌,等.维生素D对脓毒性休克导致急性呼吸窘迫综合征患者的干预价值研究[J/CD].中华危重症医学杂志(电子版),2020,13(5):321-327.
|
35 |
Koning NJ, Overmars MAH, van den Brom CE, et al. Endothelial hyperpermeability after cardiac surgery with cardiopulmonary bypass as assessed using an in vitro bioassay for endothelial barrier function[J]. Br J Anaesth, 2016, 116 (2): 223-232.
|
36 |
暴晓梅,刘畅,李发财,等.右美托咪定联合丙泊酚对感染性休克患者外周血Treg百分比及血清MDA、VCAM-1水平的影响分析[J].医药前沿,2021,11(13):56-58.
|
37 |
Joffre J, Hellman J, Ince C, et al. Endothelial responses in sepsis[J]. Am J Respir Crit Care Med, 2020, 202 (3): 361-370.
|