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中华危重症医学杂志(电子版) ›› 2022, Vol. 15 ›› Issue (02) : 155 -157. doi: 10.3877/cma.j.issn.1674-6880.2022.02.014

综述

外泌体在脓毒症心肌损伤中的研究进展
师群航1, 马莉2,()   
  1. 1. 730030 兰州,兰州大学第二临床医学院
    2. 730030 兰州,兰州大学第二医院重症医学三科
  • 收稿日期:2021-11-24 出版日期:2022-04-30
  • 通信作者: 马莉
  • 基金资助:
    兰州市人才创新创业项目(2018-RC-85)
  • Received:2021-11-24 Published:2022-04-30
引用本文:

师群航, 马莉. 外泌体在脓毒症心肌损伤中的研究进展[J]. 中华危重症医学杂志(电子版), 2022, 15(02): 155-157.

1
SingerM, DeutschmanCS, SeymourCW,et al.The third international consensus definitions for sepsis and septic shock (sepsis-3)[J].JAMA,2016,315 (8):801-810.
2
FleischmannC, ScheragA, AdhikariNK,et al.Assessment of global incidence and mortality of hospital-treated sepsis. Current estimates and limitations[J].Am J Respir Crit Care Med,2016,193 (3):259-272.
3
DenningNL, AzizM, GurienSD,et al.DAMPs and NETs in Sepsis[J].Front Immunol,2019 (10):2536.
4
VincentJL, NelsonDR, WilliamsMD.Is worsening multiple organ failure the cause of death in patients with severe sepsis?[J].Crit Care Med.2011,39 (5):1050-1055.
5
陈伟, 荣爱红, 欧珠, 等. 脓毒血症患者心率变异性和病情危重程度的关系[J].中华实用诊断与治疗杂志, 2017, 31 (6) : 541-544.
6
潘晓燕, 邓烈华. 髓系细胞触发受体-1在脓毒症心肌功能障碍中作用的研究进展[J]. 中华实用诊断与治疗杂志, 2020, 34 (3) : 318-320.
7
EssandohK, YangL, WangX,et al.Blockade of exosome generation with GW4869 dampens the sepsis-induced inflammation and cardiac dysfunction[J].Biochim Biophys Acta,2015,1852 (11):2362-2371.
8
JohnstoneRM, AdamM, HammondJR,et al.Vesicle formation during reticulocyte maturation. Association of plasma membrane activities with released vesicles (exosomes)[J].J Biol Chem,1987,262 (19):9412-9420.
9
KalluriR, LeBleuVS.The biology, function, and biomedical applications of exosomes[J].Science,2020,367 (6478):eaau6977.
10
ZhangZG, BullerB, ChoppM.Exosomes-beyond stem cells for restorative therapy in stroke and neurological injury[J].Nat Rev Neurol,2019,15 (4):193-203.
11
MuraoA, BrennerM, AzizM,et al.Exosomes in sepsis[J].Front Immunol,2020 (11):2140.
12
GaoK, JinJ, HuangC,et al.Exosomes derived from septic mouse serum modulate immune responses via exosome-associated cytokines[J].Front Immunol,2019, (10):1560.
13
TiD, HaoH, TongC,et al.LPS-preconditioned mesenchymal stromal cells modify macrophage polarization for resolution of chronic inflammation via exosome-shuttled let-7b[J].J Transl Med,2015 (13):308.
14
HerrmannIK, BertazzoS, O'CallaghanDJ,et al.Differentiating sepsis from non-infectious systemic inflammation based on microvesicle-bacteria aggregation[J].Nanoscale,2015,7 (32):13511-13520.
15
ImY, YooH, LeeJY,et al.Association of plasma exosomes with severity of organ failure and mortality in patients with sepsis[J].J Cell Mol Med,2020,24 (16):9439-9445.
16
ChoustermanBG, SwirskiFK, WeberGF.Cytokine storm and sepsis disease pathogenesis[J].Semin Immunopathol,2017,39 (5):517-528.
17
LinWC, LinCF, ChenCL,et al.Prediction of outcome in patients with acute respiratory distress syndrome by bronchoalveolar lavage inflammatory mediators[J].Exp Biol Med (Maywood),2010,235 (1):57-65.
18
FitzgeraldW, FreemanML, LedermanMM,et al.A system of cytokines encapsulated in extracellular vesicles[J].Sci Rep,2018,8 (1):8973.
19
BhatnagarS, ShinagawaK, CastellinoFJ,et al.Exosomes released from macrophages infected with intracellular pathogens stimulate a proinflammatory response in vitro and in vivo[J].Blood,2007,110 (9):3234-3244.
20
NairRR, MazzaD, BrambillaF,et al.LPS-challenged macrophages release microvesicles coated with histones[J].Front Immunol,2018 (9):1463.
21
XuJ, FengY, JeyaramA,et al.Circulating plasma extracellular vesicles from septic mice induce inflammation via microRNA- and TLR7-dependent mechanisms[J].J Immunol,2018,201 (11):3392-3400.
22
McDonaldMK, TianY, QureshiRA,et al.Functional significance of macrophage-derived exosomes in inflammation and pain[J].Pain,2014,155 (8):1527-1539.
23
SakakiH, TsukimotoM, HaradaH,et al.Autocrine regulation of macrophage activation via exocytosis of ATP and activation of P2Y11 receptor[J].PLoS One,2013,8 (4):e59778.
24
LvLL, FengY, WuM,et al.Exosomal miRNA-19b-3p of tubular epithelial cells promotes M1 macrophage activation in kidney injury[J].Cell Death Differ,2020,27 (1):210-226.
25
TungSL, BoardmanDA, SenM,et al.Regulatory T cell-derived extracellular vesicles modify dendritic cell function[J].Sci Rep,2018,8 (1):6065.
26
SeguraE, NiccoC, LombardB,et al.ICAM-1 on exosomes from mature dendritic cells is critical for efficient naive T-cell priming[J].Blood,2005,106 (1):216-223.
27
LindenberghMFS, KoerhuisDGJ, BorgEGF,et al.Bystander T-cells support clonal T-cell activation by controlling the release of dendritic cell-derived immune-stimulatory extracellular vesicles[J].Front Immunol,2019 (10):448.
28
LiuYC, YuMM, ShouST,et al.Sepsis-induced cardiomyopathy: mechanisms and treatments[J].Front Immunol,2017 (8):1021.
29
JaniszewskiM, Do CarmoAO, PedroMA,et al.Platelet-derived exosomes of septic individuals possess proapoptotic NAD(P)H oxidase activity: a novel vascular redox pathway[J].Crit Care Med,2004,32 (3):818-825.
30
AzevedoLC, JaniszewskiM, PontieriV,et al.Platelet-derived exosomes from septic shock patients induce myocardial dysfunction[J].Crit Care,2007,11 (6):R120.
31
FerdinandyP, DanialH, AmbrusI,et al.Peroxynitrite is a major contributor to cytokine-induced myocardial contractile failure[J].Circ Res,2000,87 (3):241-247.
32
GambimMH, do Carmo AdeO, MartiL,et al.Platelet-derived exosomes induce endothelial cell apoptosis through peroxynitrite generation: experimental evidence for a novel mechanism of septic vascular dysfunction[J].Crit Care,2007,11 (5):R107.
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