| 1 |
郭凤梅，邱海波. 思考与解读Sepsis新定义和诊断标准[J]. 中华内科杂志，2016，55(6):420-422.
|
| 2 |
Dellinger RP, Levy MM, Rhodes A, et al. Surviving sepsis campaign: international guidelines for management of severe sepsis and septic shock[J]. Crit Care Med, 2013, 41 (2): 580-637.
|
| 3 |
Deutschman CS, Tracey KJ. Sepsis: current dogma and new perspectives[J]. Immunity, 2014, 40 (4): 463-475.
|
| 4 |
Fisher CJ Jr, Agosti JM, Opal SM, et al. Treatment of septic shock with the tumor necrosis factor receptor: Fc fusion protein[J]. N Engl J Med, 1996, 334 (26): 1697-1702.
|
| 5 |
Zeni F, Freeman B, Natanson C. Anti-inflammatory therapies to treat sepsis and septic shock: a reassessment[J]. Crit Care Med, 1997, 25 (7): 1095-1100.
|
| 6 |
Dyugovskaya L, Polyakov A. Neutrophil apoptosis and hypoxia[J]. Fiziol Zh, 2010, 56 (5): 115-124.
|
| 7 |
Tamayo E, Gomez E, Bustamante J, et al. Evolution of neutrophil apoptosis in septic shock survivors and nonsurvivors[J]. Crit Care, 2012, 27 (4): 415. e1-e11.
|
| 8 |
Milot E, Filep JG. Regulation of neutrophil survival/apoptosis by Mcl-1[J]. Scientific World J, 2011, 11 (10): 1948-1962.
|
| 9 |
Bermejo-Martin JF, Tamayo E, Ruiz G, et al. Circula-ting neutrophil counts and mortality in septic shock[J]. Crit Care, 2014, 18 (1): 407.
|
| 10 |
Bermejo-Martin JF, Andaluz-Ojeda D, Almansa R, et al. Defining immunological dysfunction in sepsis: a requisite tool for precision medicine[J]. J Infect, 2016, 72 (5): 525-536.
|
| 11 |
邹云，李金宝. 脓毒症中树体状细胞作用及其变化的研究进展[J]. 国际麻醉学与复苏杂志，2013，6(34):568-570.
|
| 12 |
Ding Y, Chung CS, Newton S, et al. Polymicrobial sepsis induces divergent effects on splenic and peritoneal dendritic cell function in mice[J]. Shock, 2004, 22 (2): 137-144.
|
| 13 |
Flohé SB, Agrawal H, Schmitz D, et al. Dendrictic cells during polymicmbial sepsis rapidly mature but fail to initiate a protective Th1-type immune response[J]. J Leukoc Biol, 2006, 79 (3): 473-481.
|
| 14 |
Hotchkiss RS, Tinsley KW, Swanson PE, et al. Deple-tion of dendritic cells, but not macrophages, in patients with sepsis[J]. J Immunol, 2002, 168 (5): 2493-2500.
|
| 15 |
Hotchkiss RS, Monneret G, Payen D. Sepsis-induced immunosuppression: from cellular dysfunctions to immunotherapy[J]. Nat Rev Immunol, 2013, 13 (12): 862-874.
|
| 16 |
Opal SM, Laterre PF, Francois B, et al. Effect of eritoran, an antagonist of MD2-TLR4, on mortality in patients with severe sepsis: the ACCESS randomized trial[J]. JAMA, 2013, 309 (11): 1154-1162.
|
| 17 |
Adams JM, Hauser CJ, Livingston DH, et al. Early trauma polymorphonuclear neutrophil responses to chemokines are associated with development of sepsis, pneumonia, and organ failure[J]. J Trauma, 2001, 51 (3): 452-456; discussion 456-457.
|
| 18 |
曲冰杰，王宏伟，陆江阳. 改善树突状细胞功能干预性治疗脓毒症的研究进展[J]. 感染、炎症、修复，2009，10(4):239-242.
|
| 19 |
Wang HW, Yang W, Gao L, et al. Adoptive transfer of bone marrow-derived dendritic cells decreases inhibitory and regulatory T cell differentiation and improves survival in murine polymicrobial sepsis[J]. Immunol, 2015, 145 (1): 50-59.
|
| 20 |
Kockara A, Kayatas M. Renal cell apoptosis and new treatment options in sepsis-induced acute kidney injury[J]. Ren Fail, 2013, 35 (2): 291-294.
|
| 21 |
Luan YY, Dong N, Xie M, et al. The significance and regulatory mechanisms of innate immune cells in the development of sepsis[J]. Interferon Cytokine Res, 2014, 34 (1): 2-15.
|
| 22 |
Li Y, Zhao T, Liu B, et al. Inhibition of histone deacetylase 6 improves long-term survival in a lethal septic model[J]. J Trauma Acute Care Surg, 2015, 78 (2): 378-385.
|
| 23 |
Kadowaki T, Morishita A, Niki T, et al. Galectin-9 prolongs the survival of septic mice by expanding tim-3-expressing natural killer T cells and PDCA-1+ CD11c+ macrophages[J]. Crit Care, 2013, 17 (6): R284.
|
| 24 |
Inoue S, Suzuki-Utsunomiya K, Okada Y, et al. Reduction of immunocopetent T cells followed by prolonged lymphopenia in severe sepsis in the elderly[J]. Crit Care Med, 2013, 41 (3): 810-819.
|
| 25 |
Barkhausen T, Frerker C, Pütz C, et al. Depletion of NK cells in a murine polytrauma model is associated with improved outcome and a modulation of the inflammatory response[J]. Shock, 2008, 30 (4): 401-410.
|
| 26 |
Chiche L, Forel JM, Thomas G, et al. The role of natural killer cells in sepsis[J]. J Biomed Biotechnol, 2011 (2011): 986491.
|
| 27 |
Etogo AO, Nunez J, Lin CY, et al. NK but not CD1-restricted NKT cells facilitate systemic inflammation during polymicmbial intra-abdominal sepsis[J]. J Immunol, 2008, 180 (9): 6334-6345.
|
| 28 |
De Pablo R, Monserrat J, Prieto A, et al. Role of circulating lymphocytes in patients with sepsis[J]. Biomed Res Int, 2014 (2014): 671087.
|
| 29 |
Christaki E, Anyfanti P, Opal SM. Immunomodulatory therapy for sepsis: an update[J]. Expert Rev Anti Infect Ther, 2011, 9 (11): 1013-1033.
|
| 30 |
Jimbo A, Fujita E, Kouroku Y, et al. ER stress induces caspase-8 activation, stimulating cytochrome c release and caspase-9 activation[J]. Exp Cell Res, 2003, 283 (2): 156-166.
|
| 31 |
Hotchkiss RS, Swanson PE, Knudson CM, et al. Overexpression of Bcl-2 in transgenic mice decreases apoptosis and improves survival in sepsis[J]. J Immunol, 1999, 162 (7): 4148-4156.
|
| 32 |
Schwulst SJ, Muenzer JT, Peck-Palmer OM, et al. Bim siRNA decreases lymphocyte apoptosis and improves survival in sepsis[J]. Shock, 2008, 30 (2): 127-134.
|
| 33 |
Ayala A, Deol ZK, Lehman DL, et al. Polymicrobial sepsis but not low-dose endotoxin infusion causes decreased splenocyte IL-2/IFN-γ release while increasing IL-4/IL-10 production[J]. J Surg Res, 1994, 56 (6): 579-585.
|
| 34 |
Doughty L. Adaptive immune function in critical ill-ness[J]. Curr Opin Pediatr, 2016, 28 (3): 274-280.
|
| 35 |
Roth G, Moser B, Krenn C, et al. Susceptibility to programmed cell death in T-lymphocytes from septic patients: a mechanism for lymphopenia and Th2 predominance[J]. Biochem Biophys Res Commun, 2003, 308 (4): 840-846.
|
| 36 |
Venet F, Davin F, Guignant C, et al. Early assessment of leukocyte alterations at diagnosis of septic shock[J]. Shock, 2010, 34 (4): 358-363.
|
| 37 |
Monserrat J, de Pablo R, Diaz-Martin D, et al. Early alterations of B cells in patients with septic shock[J]. Crit Care, 2013, 17 (3): R105.
|
| 38 |
Kelly-Scumpia KM, Scumpia PO, Weinstein JS, et al. B cells enhance early innate immune responses during bacterial sepsis[J]. J Exp Med, 2011, 208 (8): 1673-1682.
|
| 39 |
Gommerman JL, Oh DY, Zhou X, et al. A role for CD21/CD35 and CD19 in responses to acute septic peritonitis: a potential mechanism for mast cell activation[J]. J Immunol, 2000, 165 (12): 6915-6921.
|
| 40 |
Schwulst SJ, Grayson MH, DiPasco PJ, et al. Ago-nistic monoclonal antibody against CD40 receptor decreases lymphocyte apoptosis and improves survival in sepsis[J]. J Immunol, 2006, 177 (1): 557-565.
|